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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis

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Last updated 09 junho 2024
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
The effects of sepsis on endothelium and clinical implications
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Frontiers Blood-Brain Barrier Disruption by Lipopolysaccharide
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Expression of VE-cadherin in HUVECs. A-C: VE-cadherin
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Frontiers Endothelial Damage in Sepsis: The Importance of
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
The influence of dexmedetomidine on the permeability of vascular
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Lactate induces vascular permeability via disruption of VE
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Lactate induces vascular permeability via disruption of VE
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Endothelial barrier dysfunction in systemic inflammation is
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Antioxidants, Free Full-Text
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
PLCβ2 Promotes VEGF-Induced Vascular Permeability
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Vascular permeability in retinopathy is regulated by VEGFR2 Y949
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
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